Verdile, G <br/><br/>
Baculoviruses expressing the human familial Alzheimer's disease presenilin 1 mutation lacking exon 9 increase levels of an amyloid beta-like protein in Sf9 cells.  [electronic resource] 

 -  Molecular psychiatry  Jun 2004 
 - 594-602 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S. 
<br/><br/><label>ISSN: </label>1359-4184 
<br/><br/><label>Standard No.: </label>10.1038/sj.mp.4001458  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Alzheimer Disease--genetics<br/>Amyloid beta-Peptides--genetics<br/>Animals<br/>Baculoviridae--genetics<br/>Cell Line<br/>Exons--genetics<br/>Humans<br/>Membrane Proteins--genetics<br/>Presenilin-1<br/>Recombinant Proteins--metabolism<br/>Sequence Deletion<br/>Spodoptera<br/>Transfection