Karray, Saoussen <br/><br/>
Complete loss of Fas ligand gene causes massive lymphoproliferation and early death, indicating a residual activity of gld allele.  [electronic resource] 

 -  Journal of immunology (Baltimore, Md. : 1950)  Feb 2004 
 - 2118-25 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0022-1767 
<br/><br/><label>Standard No.: </label>10.4049/jimmunol.172.4.2118  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Alleles<br/>Animals<br/>Autoantibodies--blood<br/>CD3 Complex--biosynthesis<br/>CD4 Antigens--metabolism<br/>CD8 Antigens--metabolism<br/>Cell Movement--genetics<br/>Crosses, Genetic<br/>Fas Ligand Protein<br/>Female<br/>Gene Deletion<br/>Gene Silencing--immunology<br/>Glomerulonephritis--genetics<br/>Hypergammaglobulinemia--genetics<br/>Leukocyte Common Antigens--biosynthesis<br/>Ligands<br/>Liver--immunology<br/>Lymphoproliferative Disorders--genetics<br/>Male<br/>Membrane Glycoproteins--deficiency<br/>Mice<br/>Mice, Inbred C57BL<br/>Mice, Knockout<br/>Mice, Mutant Strains<br/>Mice, Transgenic<br/>Salivary Glands--immunology<br/>T-Lymphocyte Subsets--immunology<br/>fas Receptor--metabolism