Loufrani, Laurent <br/><br/>
Absence of dystrophin in mice reduces NO-dependent vascular function and vascular density: total recovery after a treatment with the aminoglycoside gentamicin.  [electronic resource] 

 -  Arteriosclerosis, thrombosis, and vascular biology  Apr 2004 
 - 671-6 p.  digital 
<br/><br/> Publication Type: Comparative Study; Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1524-4636 
<br/><br/><label>Standard No.: </label>10.1161/01.ATV.0000118683.99628.42  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Carotid Arteries--drug effects<br/>Codon, Nonsense<br/>Coronary Vessels--pathology<br/>Dystrophin--deficiency<br/>Endothelium, Vascular--drug effects<br/>Gene Expression Regulation--drug effects<br/>Gentamicins--pharmacology<br/>Hemorheology<br/>Male<br/>Mesenteric Arteries--drug effects<br/>Mice<br/>Mice, Inbred C57BL<br/>Mice, Inbred mdx<br/>Mice, Knockout<br/>Muscle, Skeletal--blood supply<br/>NG-Nitroarginine Methyl Ester--pharmacology<br/>Nitric Oxide--physiology<br/>Nitric Oxide Synthase--biosynthesis<br/>Nitric Oxide Synthase Type II<br/>Nitric Oxide Synthase Type III<br/>Signal Transduction<br/>Stress, Mechanical<br/>Vasoconstriction--drug effects<br/>Vasoconstrictor Agents--pharmacology<br/>Vimentin--deficiency