Song, Ye <br/><br/>
Fluvastatin prevents nephropathy likely through suppression of connective tissue growth factor-mediated extracellular matrix accumulation.  [electronic resource] 

 -  Experimental and molecular pathology  Feb 2004 
 - 66-75 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0014-4800 
<br/><br/><label>Standard No.: </label>10.1016/j.yexmp.2003.08.002  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Anticholesteremic Agents--therapeutic use<br/>Blood Urea Nitrogen<br/>Connective Tissue Growth Factor<br/>Creatinine--urine<br/>Disease Models, Animal<br/>Extracellular Matrix--metabolism<br/>Fatty Acids, Monounsaturated--therapeutic use<br/>Fluvastatin<br/>Immediate-Early Proteins--biosynthesis<br/>Indoles--therapeutic use<br/>Intercellular Signaling Peptides and Proteins--biosynthesis<br/>Kidney Failure, Chronic--metabolism<br/>Kidney Glomerulus--drug effects<br/>Male<br/>Matrix Metalloproteinase Inhibitors<br/>Nephrectomy<br/>Proteinuria<br/>RNA, Messenger--metabolism<br/>Rats<br/>Rats, Wistar<br/>Tissue Inhibitor of Metalloproteinase-2--biosynthesis<br/>Up-Regulation