Molecular and functional consequences of Smad4 C-terminal missense mutations in colorectal tumour cells. [electronic resource]
- The Biochemical journal Apr 2004
- 209-16 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
1470-8728
10.1042/BJ20031886 doi
ADP-Ribosylation Factor 1--metabolism Active Transport, Cell Nucleus Amino Acid Substitution Cell Division--drug effects Cell Nucleus--metabolism Colorectal Neoplasms--metabolism DNA-Binding Proteins--chemistry Dimerization Forkhead Transcription Factors Gene Expression Regulation, Neoplastic Humans Macromolecular Substances Models, Molecular Mutation, Missense Neoplasm Proteins--chemistry Nerve Growth Factors Point Mutation Protein Binding Protein Conformation Protein Structure, Tertiary Signal Transduction Smad Proteins Smad2 Protein Smad3 Protein Smad4 Protein Structure-Activity Relationship Trans-Activators--chemistry Transcription Factors--metabolism Transcription, Genetic Transforming Growth Factor beta--pharmacology Xenopus Proteins