Hermel, E <br/><br/>
Specific caspase interactions and amplification are involved in selective neuronal vulnerability in Huntington's disease.  [electronic resource] 

 -  Cell death and differentiation  Apr 2004 
 - 424-38 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S. 
<br/><br/><label>ISSN: </label>1350-9047 
<br/><br/><label>Standard No.: </label>10.1038/sj.cdd.4401358  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Brain--metabolism<br/>Caspase 2<br/>Caspase 3<br/>Caspase 6<br/>Caspase 7<br/>Caspases--metabolism<br/>Cell Death--physiology<br/>Cerebral Cortex--metabolism<br/>Corpus Striatum--metabolism<br/>Disease Models, Animal<br/>Gene Expression Regulation--genetics<br/>Humans<br/>Huntingtin Protein<br/>Huntington Disease--genetics<br/>Mice<br/>Mice, Transgenic--genetics<br/>Mutation<br/>Nerve Tissue Proteins--genetics<br/>Neurons--metabolism<br/>Nuclear Proteins--genetics