Hansen, Jason M <br/><br/>
A novel hypothesis for thalidomide-induced limb teratogenesis: redox misregulation of the NF-kappaB pathway.  [electronic resource] 

 -  Antioxidants & redox signaling  Feb 2004 
 - 1-14 p.  digital 
<br/><br/> Publication Type: Editorial; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S. 
<br/><br/><label>ISSN: </label>1523-0864 
<br/><br/><label>Standard No.: </label>10.1089/152308604771978291  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>DNA--metabolism<br/>Free Radicals--metabolism<br/>Gene Expression Regulation, Developmental--drug effects<br/>Glutathione--metabolism<br/>In Situ Hybridization<br/>Limb Buds--drug effects<br/>Limb Deformities, Congenital--chemically induced<br/>Models, Biological<br/>NF-kappa B--metabolism<br/>Oxidation-Reduction--drug effects<br/>Oxidative Stress--physiology<br/>Rabbits<br/>Rats<br/>Signal Transduction--drug effects<br/>Species Specificity<br/>Teratogens--metabolism<br/>Thalidomide--metabolism