Wang, Wei <br/><br/>
The susceptibility to experimental myasthenia gravis of STAT6-/- and STAT4-/- BALB/c mice suggests a pathogenic role of Th1 cells.  [electronic resource] 

 -  Journal of immunology (Baltimore, Md. : 1950)  Jan 2004 
 - 97-103 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, U.S. Gov't, P.H.S. 
<br/><br/><label>ISSN: </label>0022-1767 
<br/><br/><label>Standard No.: </label>10.4049/jimmunol.172.1.97  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Autoantibodies--biosynthesis<br/>CD4-Positive T-Lymphocytes--cytology<br/>Cell Division--immunology<br/>DNA-Binding Proteins--deficiency<br/>Female<br/>Genetic Predisposition to Disease<br/>Immunoglobulin G--biosynthesis<br/>Interferon-gamma--metabolism<br/>Interleukin-4--metabolism<br/>Mice<br/>Mice, Inbred BALB C<br/>Mice, Knockout<br/>Myasthenia Gravis, Autoimmune, Experimental--genetics<br/>Peptide Fragments--pharmacology<br/>Protein Subunits--pharmacology<br/>Receptors, Nicotinic--immunology<br/>STAT4 Transcription Factor<br/>STAT6 Transcription Factor<br/>Signal Transduction--genetics<br/>Th1 Cells--immunology<br/>Torpedo--immunology<br/>Trans-Activators--deficiency