Lynch, Nicholas J <br/><br/>
Microglial activation and increased synthesis of complement component C1q precedes blood-brain barrier dysfunction in rats.  [electronic resource] 

 -  Molecular immunology  Jan 2004 
 - 709-16 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0161-5890 
<br/><br/><label>Standard No.: </label>10.1016/j.molimm.2003.08.009  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Base Sequence<br/>Blood-Brain Barrier--drug effects<br/>Brain--drug effects<br/>Complement C1q--biosynthesis<br/>DNA, Complementary--genetics<br/>DNA-Binding Proteins--genetics<br/>Early Growth Response Protein 1<br/>Immediate-Early Proteins<br/>In Situ Hybridization<br/>Interleukin-1--genetics<br/>Male<br/>Microglia--drug effects<br/>Models, Neurological<br/>Neurotoxins--toxicity<br/>RNA, Messenger--genetics<br/>Rats<br/>Rats, Inbred F344<br/>Transcription Factors--genetics<br/>Tumor Necrosis Factor-alpha--genetics<br/>alpha-Chlorohydrin--toxicity