Beggs, Hilary E
FAK deficiency in cells contributing to the basal lamina results in cortical abnormalities resembling congenital muscular dystrophies. [electronic resource]
- Neuron Oct 2003
- 501-14 p. digital
Publication Type: Comparative Study; Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
ISSN: 0896-6273
Standard No.: 10.1016/s0896-6273(03)00666-4 doi
Subjects--Topical Terms: Animals Astrocytes--metabolism Bacterial Proteins--metabolism Basement Membrane--metabolism Blotting, Western Calbindin 2 Calbindins Carrier Proteins--metabolism Cell Adhesion Molecules, Neuronal--metabolism Cells, Cultured Cerebral Cortex--abnormalities Cytoskeletal Proteins--metabolism DNA-Binding Proteins--metabolism Disease Models, Animal Dura Mater Dystroglycans Embryo, Mammalian Extracellular Matrix Proteins--metabolism Fibroblasts--metabolism Focal Adhesion Kinase 1 Focal Adhesion Protein-Tyrosine Kinases Glial Fibrillary Acidic Protein--metabolism Heterozygote Homeodomain Proteins--metabolism Immunohistochemistry Infections Intracellular Signaling Peptides and Proteins Lamins--metabolism Membrane Glycoproteins--metabolism Mice Mice, Knockout Microscopy, Electron Microtubule-Associated Proteins--metabolism Muscular Dystrophies--congenital Mutation Nerve Tissue Proteins Neurons--metabolism Otx Transcription Factors Phosphopyruvate Hydratase--metabolism Phosphotyrosine--metabolism Precipitin Tests Protein-Tyrosine Kinases--deficiency Reelin Protein S100 Calcium Binding Protein G--metabolism Serine Endopeptidases Silver Staining Staining and Labeling Transcription Factors--metabolism src-Family Kinases--metabolism