Rajasekhar, Vinagolu K <br/><br/>
Oncogenic Ras and Akt signaling contribute to glioblastoma formation by differential recruitment of existing mRNAs to polysomes.  [electronic resource] 

 -  Molecular cell  Oct 2003 
 - 889-901 p.  digital 
<br/><br/> Publication Type: Comparative Study; Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S. 
<br/><br/><label>ISSN: </label>1097-2765 
<br/><br/><label>Standard No.: </label>10.1016/s1097-2765(03)00395-2  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Brain Neoplasms--genetics<br/>Cell Cycle Proteins--metabolism<br/>Cell Transformation, Neoplastic--drug effects<br/>Cluster Analysis<br/>Enzyme Inhibitors--pharmacology<br/>Gene Expression Regulation, Neoplastic--genetics<br/>Glioblastoma--genetics<br/>Humans<br/>Mice<br/>Mice, Transgenic<br/>Oligonucleotide Array Sequence Analysis<br/>Polyribosomes--genetics<br/>Protein Biosynthesis--drug effects<br/>Protein Serine-Threonine Kinases<br/>Proto-Oncogene Proteins--antagonists & inhibitors<br/>Proto-Oncogene Proteins c-akt<br/>RNA, Messenger--analysis<br/>Signal Transduction--drug effects<br/>Tumor Cells, Cultured<br/>ras Proteins--antagonists & inhibitors