Debnath, Jayanta <br/><br/>
Akt activation disrupts mammary acinar architecture and enhances proliferation in an mTOR-dependent manner.  [electronic resource] 

 -  The Journal of cell biology  Oct 2003 
 - 315-26 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.; Research Support, U.S. Gov't, P.H.S. 
<br/><br/><label>ISSN: </label>0021-9525 
<br/><br/><label>Standard No.: </label>10.1083/jcb.200304159  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Antibiotics, Antineoplastic--pharmacology<br/>Apoptosis<br/>Breast--cytology<br/>Cell Division--drug effects<br/>Cell Line<br/>Cell Line, Transformed<br/>Cell Polarity--drug effects<br/>Cell Size--drug effects<br/>Cell Survival--drug effects<br/>Cyclin D1--metabolism<br/>DNA--analysis<br/>Dose-Response Relationship, Drug<br/>Enzyme Activation<br/>Epithelial Cells--cytology<br/>Humans<br/>Morphogenesis--drug effects<br/>Protein Kinase Inhibitors<br/>Protein Kinases--drug effects<br/>Protein Serine-Threonine Kinases<br/>Proto-Oncogene Proteins--genetics<br/>Proto-Oncogene Proteins c-akt<br/>Retroviridae--genetics<br/>Sirolimus--pharmacology<br/>TOR Serine-Threonine Kinases