MacLean, Helen E <br/><br/>
Absence of transcription factor c-maf causes abnormal terminal differentiation of hypertrophic chondrocytes during endochondral bone development.  [electronic resource] 

 -  Developmental biology  Oct 2003 
 - 51-63 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S. 
<br/><br/><label>ISSN: </label>0012-1606 
<br/><br/><label>Standard No.: </label>10.1016/s0012-1606(03)00324-5  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Apoptosis<br/>Bone Development--physiology<br/>Cell Differentiation--physiology<br/>Cell Division<br/>Chondrocytes--cytology<br/>Collagenases--genetics<br/>DNA-Binding Proteins--physiology<br/>Hypertrophy<br/>Matrix Metalloproteinase 13<br/>Mice<br/>Mice, Inbred BALB C<br/>Osteoblasts--physiology<br/>Proto-Oncogene Proteins--physiology<br/>Proto-Oncogene Proteins c-maf<br/>Tumor Suppressor Protein p53--analysis