Time course of early motor and neuropathological anomalies in a knock-in mouse model of Huntington's disease with 140 CAG repeats. [electronic resource]
- The Journal of comparative neurology Oct 2003
- 11-26 p. digital
Publication Type: Journal Article
0021-9967
10.1002/cne.10776 doi
Animals Brain--metabolism Cerebral Cortex--metabolism Corpus Striatum--metabolism Disease Models, Animal Female Grooming--physiology Huntingtin Protein Huntington Disease--genetics Immunohistochemistry Inclusion Bodies--genetics Lameness, Animal--genetics Male Mice Mice, Transgenic Motor Activity--genetics Nerve Tissue Proteins--deficiency Neurons--metabolism Neuropil--metabolism Nuclear Proteins--deficiency Nucleus Accumbens--metabolism Olfactory Pathways--metabolism Recombinant Fusion Proteins--genetics Time Factors Trinucleotide Repeats--genetics