Oxidative stress induces nuclear loss of DNA repair proteins Ku70 and Ku80 and apoptosis in pancreatic acinar AR42J cells. [electronic resource]
- The Journal of biological chemistry Sep 2003
- 36676-87 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
0021-9258
10.1074/jbc.M303692200 doi
Antigens, Nuclear--biosynthesis Apoptosis Blotting, Western Caspase 3 Caspase Inhibitors Cell Death Cell Line, Tumor Cell Nucleus--metabolism Cell Survival Cytoplasm--metabolism DNA Fragmentation DNA Helicases DNA Repair DNA, Complementary--metabolism DNA-Binding Proteins--biosynthesis Dose-Response Relationship, Drug Enzyme Inhibitors--pharmacology Genes, Dominant Glucose Oxidase--metabolism Glutathione Transferase--metabolism Humans Hydrogen Peroxide--pharmacology Ku Autoantigen Microscopy, Fluorescence Oxidative Stress Pancreas--cytology Plasmids--metabolism Protein Binding Proto-Oncogene Proteins--metabolism Proto-Oncogene Proteins c-bcl-2--metabolism Recombinant Fusion Proteins--metabolism Time Factors Transfection Tumor Suppressor Protein p53--metabolism alpha Karyopherins--chemistry bcl-2-Associated X Protein beta Karyopherins--metabolism