Kitazawa, M <br/><br/>
Dieldrin induces apoptosis by promoting caspase-3-dependent proteolytic cleavage of protein kinase Cdelta in dopaminergic cells: relevance to oxidative stress and dopaminergic degeneration.  [electronic resource] 

 -  Neuroscience  2003 
 - 945-64 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, U.S. Gov't, P.H.S. 
<br/><br/><label>ISSN: </label>0306-4522 
<br/><br/><label>Standard No.: </label>10.1016/s0306-4522(03)00226-4  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Apoptosis--drug effects<br/>Caspase 3<br/>Caspases--drug effects<br/>Cytochrome c Group--metabolism<br/>Dieldrin--pharmacology<br/>Dopamine--metabolism<br/>Dose-Response Relationship, Drug<br/>Enzyme Inhibitors--pharmacology<br/>Feedback, Physiological--drug effects<br/>Free Radical Scavengers--pharmacology<br/>Gene Expression Regulation, Enzymologic--drug effects<br/>Metalloporphyrins--pharmacology<br/>Neurons--drug effects<br/>Oxidative Stress--drug effects<br/>PC12 Cells<br/>Parkinsonian Disorders--enzymology<br/>Peptide Hydrolases--drug effects<br/>Protein Kinase C--genetics<br/>Protein Kinase C-delta<br/>Protein Subunits--pharmacology<br/>Rats<br/>Reaction Time--drug effects<br/>Superoxide Dismutase--metabolism