Atarashi, Ryuichiro <br/><br/>
Deletion of N-terminal residues 23-88 from prion protein (PrP) abrogates the potential to rescue PrP-deficient mice from PrP-like protein/doppel-induced Neurodegeneration.  [electronic resource] 

 -  The Journal of biological chemistry  Aug 2003 
 - 28944-9 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0021-9258 
<br/><br/><label>Standard No.: </label>10.1074/jbc.M303655200  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Ataxia--genetics<br/>Brain--enzymology<br/>Calbindins<br/>Cell Death--genetics<br/>Cricetinae<br/>Detergents<br/>GPI-Linked Proteins<br/>Gene Deletion<br/>Gene Expression<br/>Glutathione Peroxidase--metabolism<br/>Mice<br/>Mice, Transgenic<br/>Nerve Degeneration--chemically induced<br/>Neurons--metabolism<br/>Peptide Fragments--chemistry<br/>Phenotype<br/>Pregnancy Proteins--chemistry<br/>Prions--genetics<br/>Purkinje Cells--pathology<br/>Repetitive Sequences, Nucleic Acid<br/>S100 Calcium Binding Protein G--analysis<br/>Solubility<br/>Superoxide Dismutase--metabolism