Cai, Ling <br/><br/>
The tumor-selective over-expression of the human Hsp70 gene is attributed to the aberrant controls at both initiation and elongation levels of transcription.  [electronic resource] 

 -  Cell research  Apr 2003 
 - 93-109 p.  digital 
<br/><br/> Publication Type: Comparative Study; Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1001-0602 
<br/><br/><label>Standard No.: </label>10.1038/sj.cr.7290154  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>5' Untranslated Regions--genetics<br/>Base Sequence--genetics<br/>Cell Transformation, Neoplastic--genetics<br/>Gene Expression Regulation, Neoplastic--genetics<br/>Genes, Regulator--genetics<br/>HSP70 Heat-Shock Proteins--biosynthesis<br/>Humans<br/>Mutation--genetics<br/>Neoplasms--genetics<br/>Promoter Regions, Genetic--genetics<br/>Protein Biosynthesis--genetics<br/>RNA, Messenger--biosynthesis<br/>Transcription Initiation Site--physiology<br/>Transcriptional Elongation Factors--genetics<br/>Tumor Cells, Cultured<br/>Tumor Suppressor Protein p53--deficiency