Shibata, M <br/><br/>
Activation of caspase-12 by endoplasmic reticulum stress induced by transient middle cerebral artery occlusion in mice.  [electronic resource] 

 -  Neuroscience  2003 
 - 491-9 p.  digital 
<br/><br/> Publication Type: Comparative Study; Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0306-4522 
<br/><br/><label>Standard No.: </label>10.1016/s0306-4522(02)00910-7  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Apoptosis--physiology<br/>Carrier Proteins--metabolism<br/>Caspase 12<br/>Caspase 7<br/>Caspases--metabolism<br/>Cerebral Cortex--metabolism<br/>Corpus Striatum--metabolism<br/>Cytosol--metabolism<br/>DNA Fragmentation<br/>Endoplasmic Reticulum--enzymology<br/>Endoplasmic Reticulum Chaperone BiP<br/>Enzyme Activation<br/>HSP70 Heat-Shock Proteins--metabolism<br/>Heat-Shock Proteins<br/>Immunohistochemistry--methods<br/>In Situ Nick-End Labeling--methods<br/>Infarction, Middle Cerebral Artery--complications<br/>Ischemic Attack, Transient--etiology<br/>Male<br/>Membrane Proteins--metabolism<br/>Mice<br/>Mice, Inbred C57BL<br/>Microsomes--metabolism<br/>Molecular Chaperones--metabolism<br/>Nerve Tissue Proteins--metabolism<br/>Nuclear Proteins--metabolism<br/>Reperfusion<br/>Signal Transduction--physiology<br/>Stress, Physiological--etiology<br/>Subcellular Fractions--chemistry<br/>Time Factors