PIP(2) activates KCNQ channels, and its hydrolysis underlies receptor-mediated inhibition of M currents. [electronic resource]
- Neuron Mar 2003
- 963-75 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.; Research Support, U.S. Gov't, P.H.S.
0896-6273
10.1016/s0896-6273(03)00125-9 doi
Androstadienes--pharmacology Animals Bradykinin--pharmacology CHO Cells Cells, Cultured Cricetinae Electric Conductivity Enzyme Inhibitors--pharmacology Female Hydrolysis KCNQ2 Potassium Channel KCNQ3 Potassium Channel Microscopy, Confocal Mutation Neurons--physiology Patch-Clamp Techniques Phosphatidylinositol 4,5-Diphosphate--metabolism Phosphoinositide-3 Kinase Inhibitors Potassium Channels--genetics Potassium Channels, Voltage-Gated Rats Rats, Sprague-Dawley Receptor, Muscarinic M1 Receptors, Muscarinic--physiology Recombinant Proteins Second Messenger Systems Signal Transduction Superior Cervical Ganglion--cytology Tetraethylammonium--pharmacology Transfection Wortmannin Xenopus laevis