Zhang, Hailin

PIP(2) activates KCNQ channels, and its hydrolysis underlies receptor-mediated inhibition of M currents. [electronic resource] - Neuron Mar 2003 - 963-75 p. digital

Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.; Research Support, U.S. Gov't, P.H.S.

0896-6273

10.1016/s0896-6273(03)00125-9 doi


Androstadienes--pharmacology
Animals
Bradykinin--pharmacology
CHO Cells
Cells, Cultured
Cricetinae
Electric Conductivity
Enzyme Inhibitors--pharmacology
Female
Hydrolysis
KCNQ2 Potassium Channel
KCNQ3 Potassium Channel
Microscopy, Confocal
Mutation
Neurons--physiology
Patch-Clamp Techniques
Phosphatidylinositol 4,5-Diphosphate--metabolism
Phosphoinositide-3 Kinase Inhibitors
Potassium Channels--genetics
Potassium Channels, Voltage-Gated
Rats
Rats, Sprague-Dawley
Receptor, Muscarinic M1
Receptors, Muscarinic--physiology
Recombinant Proteins
Second Messenger Systems
Signal Transduction
Superior Cervical Ganglion--cytology
Tetraethylammonium--pharmacology
Transfection
Wortmannin
Xenopus laevis