Li, Yuekui <br/><br/>
Interleukin-1 mediates pathological effects of microglia on tau phosphorylation and on synaptophysin synthesis in cortical neurons through a p38-MAPK pathway.  [electronic resource] 

 -  The Journal of neuroscience : the official journal of the Society for Neuroscience  Mar 2003 
 - 1605-11 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S. 
<br/><br/><label>ISSN: </label>1529-2401 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Amyloid beta-Protein Precursor--pharmacology<br/>Animals<br/>Cell Communication--physiology<br/>Cells, Cultured<br/>Cerebral Cortex--cytology<br/>Coculture Techniques<br/>Interleukin 1 Receptor Antagonist Protein<br/>Interleukin-1--genetics<br/>Lipopolysaccharides--pharmacology<br/>Mice<br/>Microglia--cytology<br/>Mitogen-Activated Protein Kinases--metabolism<br/>Neurons--cytology<br/>Neuroprotective Agents--pharmacology<br/>Phosphorylation--drug effects<br/>Rats<br/>Rats, Sprague-Dawley<br/>Sialoglycoproteins--pharmacology<br/>Signal Transduction--drug effects<br/>Synaptophysin--metabolism<br/>Tumor Necrosis Factor-alpha--antagonists & inhibitors<br/>p38 Mitogen-Activated Protein Kinases<br/>tau Proteins--metabolism