Roca, Ramon <br/><br/>
Loss of p16INK4a results in increased glucocorticoid receptor activity during fibrosarcoma development.  [electronic resource] 

 -  Proceedings of the National Academy of Sciences of the United States of America  Mar 2003 
 - 3113-8 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0027-8424 
<br/><br/><label>Standard No.: </label>10.1073/pnas.0634912100  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Base Sequence<br/>Cell Division--drug effects<br/>Cyclin-Dependent Kinase Inhibitor p16--deficiency<br/>DNA Methylation<br/>DNA, Neoplasm--chemistry<br/>Dexamethasone--pharmacology<br/>Fibrosarcoma--etiology<br/>Genes, p16<br/>Mice<br/>Precancerous Conditions--etiology<br/>Receptors, Glucocorticoid--genetics<br/>Transcriptional Activation<br/>Tumor Cells, Cultured