Serreze, David V <br/><br/>
Paralytic autoimmune myositis develops in nonobese diabetic mice made Th1 cytokine-deficient by expression of an IFN-gamma receptor beta-chain transgene.  [electronic resource] 

 -  Journal of immunology (Baltimore, Md. : 1950)  Mar 2003 
 - 2742-9 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S. 
<br/><br/><label>ISSN: </label>0022-1767 
<br/><br/><label>Standard No.: </label>10.4049/jimmunol.170.5.2742  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Adoptive Transfer<br/>Animals<br/>CD8-Positive T-Lymphocytes--immunology<br/>Cell Movement--genetics<br/>Cytokines--deficiency<br/>Diabetes Mellitus, Type 1--genetics<br/>Female<br/>Gene Expression Regulation--immunology<br/>Interferon-gamma--antagonists & inhibitors<br/>Kinetics<br/>Macrophages--pathology<br/>Mice<br/>Mice, Inbred C57BL<br/>Mice, Inbred NOD<br/>Mice, SCID<br/>Mice, Transgenic<br/>Nervous System Autoimmune Disease, Experimental--genetics<br/>Paralysis--genetics<br/>Receptors, Interferon--biosynthesis<br/>Sex Characteristics<br/>Spleen--immunology<br/>Th1 Cells--immunology<br/>Transgenes--immunology<br/>Tumor Necrosis Factor-alpha--antagonists & inhibitors<br/>Interferon gamma Receptor