Fenton, James A L <br/><br/>
Genomic characterization of the chromosomal breakpoints of t(4;14) of multiple myeloma suggests more than one possible aetiological mechanism.  [electronic resource] 

 -  Oncogene  Feb 2003 
 - 1103-13 p.  digital 
<br/><br/> Publication Type: Comparative Study; Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0950-9232 
<br/><br/><label>Standard No.: </label>10.1038/sj.onc.1206335  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>B-Lymphocytes--pathology<br/>Base Sequence<br/>Cell Transformation, Neoplastic--genetics<br/>Chromosome Breakage<br/>Chromosomes, Human, Pair 14--genetics<br/>Chromosomes, Human, Pair 4--genetics<br/>Clone Cells--pathology<br/>Gene Rearrangement, B-Lymphocyte, Heavy Chain<br/>Genes, Immunoglobulin<br/>Genes, Switch<br/>Humans<br/>Immunoglobulin Class Switching--genetics<br/>Immunoglobulin Switch Region--genetics<br/>Immunoglobulin mu-Chains--genetics<br/>Models, Biological<br/>Molecular Sequence Data<br/>Multiple Myeloma--etiology<br/>Neoplastic Stem Cells--pathology<br/>Polymerase Chain Reaction--methods<br/>Sequence Alignment<br/>Sequence Homology, Nucleic Acid<br/>Translocation, Genetic--genetics