Muņoz-Pinedo, Cristina <br/><br/>
Inhibition of glucose metabolism sensitizes tumor cells to death receptor-triggered apoptosis through enhancement of death-inducing signaling complex formation and apical procaspase-8 processing.  [electronic resource] 

 -  The Journal of biological chemistry  Apr 2003 
 - 12759-68 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0021-9258 
<br/><br/><label>Standard No.: </label>10.1074/jbc.M212392200  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Adenosine Triphosphate--metabolism<br/>Apoptosis--drug effects<br/>Apoptosis Regulatory Proteins<br/>Breast Neoplasms<br/>Caspase 8<br/>Caspase 9<br/>Caspases--metabolism<br/>Cytochrome c Group--metabolism<br/>Deoxyglucose--metabolism<br/>Enzyme Activation<br/>Enzyme Precursors--metabolism<br/>Female<br/>Glucose--antagonists & inhibitors<br/>HeLa Cells<br/>Humans<br/>Kinetics<br/>Lymphoma<br/>Membrane Glycoproteins--physiology<br/>Mitochondria--physiology<br/>Protein Processing, Post-Translational<br/>Pyruvates--metabolism<br/>Signal Transduction--physiology<br/>TNF-Related Apoptosis-Inducing Ligand<br/>Tumor Cells, Cultured<br/>Tumor Necrosis Factor-alpha--pharmacology<br/>U937 Cells<br/>fas Receptor--physiology