Wolvetang, E J <br/><br/>
ETS2 overexpression in transgenic models and in Down syndrome predisposes to apoptosis via the p53 pathway.  [electronic resource] 

 -  Human molecular genetics  Feb 2003 
 - 247-55 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0964-6906 
<br/><br/><label>Standard No.: </label>10.1093/hmg/ddg015  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Apoptosis--genetics<br/>DNA-Binding Proteins<br/>Down Syndrome--metabolism<br/>Gene Expression Regulation--genetics<br/>HeLa Cells<br/>Humans<br/>Mice<br/>Mice, Transgenic<br/>Proto-Oncogene Protein c-ets-2<br/>Proto-Oncogene Proteins--biosynthesis<br/>Repressor Proteins<br/>Thymus Gland--pathology<br/>Trans-Activators--biosynthesis<br/>Transcription Factors<br/>Tumor Suppressor Protein p53--genetics