Zhong, Bin <br/><br/>
Human neutrophils utilize a Rac/Cdc42-dependent MAPK pathway to direct intracellular granule mobilization toward ingested microbial pathogens.  [electronic resource] 

 -  Blood  Apr 2003 
 - 3240-8 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S. 
<br/><br/><label>ISSN: </label>0006-4971 
<br/><br/><label>Standard No.: </label>10.1182/blood-2001-12-0180  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Candida albicans<br/>Cytoplasmic Granules--physiology<br/>Enzyme Activation--drug effects<br/>Enzyme Inhibitors--pharmacology<br/>Flavonoids--pharmacology<br/>Genes, Dominant<br/>Humans<br/>MAP Kinase Kinase 1<br/>MAP Kinase Signaling System--drug effects<br/>Mitogen-Activated Protein Kinase 1--metabolism<br/>Mitogen-Activated Protein Kinase 3<br/>Mitogen-Activated Protein Kinase Kinases--antagonists & inhibitors<br/>Mitogen-Activated Protein Kinases--metabolism<br/>Mutagenesis, Site-Directed<br/>Neutrophils--physiology<br/>Phagocytosis--physiology<br/>Phosphorylation<br/>Protein Processing, Post-Translational<br/>Protein Serine-Threonine Kinases--antagonists & inhibitors<br/>Recombinant Fusion Proteins--antagonists & inhibitors<br/>Transduction, Genetic<br/>cdc42 GTP-Binding Protein--genetics<br/>p21-Activated Kinases<br/>rac1 GTP-Binding Protein--genetics<br/>rho GTP-Binding Proteins--genetics