Kerkvliet, Nancy I <br/><br/>
T lymphocytes are direct, aryl hydrocarbon receptor (AhR)-dependent targets of 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD): AhR expression in both CD4+ and CD8+ T cells is necessary for full suppression of a cytotoxic T lymphocyte response by TCDD.  [electronic resource] 

 -  Toxicology and applied pharmacology  Dec 2002 
 - 146-52 p.  digital 
<br/><br/> Publication Type: Comparative Study; Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S. 
<br/><br/><label>ISSN: </label>0041-008X 
<br/><br/><label>Standard No.: </label>10.1006/taap.2002.9537  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Body Weight<br/>CD4-Positive T-Lymphocytes--drug effects<br/>CD8-Positive T-Lymphocytes--drug effects<br/>Cytotoxicity Tests, Immunologic<br/>Environmental Pollutants--toxicity<br/>Female<br/>Host vs Graft Reaction--immunology<br/>Lymphocyte Activation--drug effects<br/>Male<br/>Mice<br/>Mice, Inbred C57BL<br/>Mice, Inbred DBA<br/>Mice, Knockout<br/>Polychlorinated Dibenzodioxins--toxicity<br/>Receptors, Aryl Hydrocarbon--immunology<br/>T-Lymphocytes, Cytotoxic--drug effects