Aliev, Gjumrakch <br/><br/>
Atherosclerotic lesions and mitochondria DNA deletions in brain microvessels as a central target for the development of human AD and AD-like pathology in aged transgenic mice.  [electronic resource] 

 -  Annals of the New York Academy of Sciences  Nov 2002 
 - 45-64 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S. 
<br/><br/><label>ISSN: </label>0077-8923 
<br/><br/><label>Standard No.: </label>10.1111/j.1749-6632.2002.tb04798.x  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Alzheimer Disease--genetics<br/>Amyloid beta-Protein Precursor--genetics<br/>Animals<br/>Arteriosclerosis--pathology<br/>Base Sequence<br/>Cerebrovascular Disorders--pathology<br/>DNA Probes<br/>DNA, Mitochondrial--genetics<br/>Humans<br/>Mice<br/>Mice, Transgenic<br/>Microcirculation--pathology<br/>Models, Animal<br/>Oxidative Stress<br/>Sequence Deletion