Mizuki, Masao

Suppression of myeloid transcription factors and induction of STAT response genes by AML-specific Flt3 mutations. [electronic resource] - Blood Apr 2003 - 3164-73 p. digital

Publication Type: Journal Article; Research Support, Non-U.S. Gov't

0006-4971

10.1182/blood-2002-06-1677 doi


Acute Disease
Animals
CCAAT-Enhancer-Binding Protein-alpha--biosynthesis
Cell Line--metabolism
Cluster Analysis
Computer Systems
Gene Expression Profiling
Gene Expression Regulation
Gene Expression Regulation, Leukemic
Genes, Synthetic
Hematopoietic Stem Cells--metabolism
Humans
Interleukin-3--pharmacology
Leukemia, Myeloid--genetics
Mice
Myeloid Cells--metabolism
Neoplasm Proteins--biosynthesis
Oligonucleotide Array Sequence Analysis
Protein Isoforms--physiology
Protein Serine-Threonine Kinases
Proto-Oncogene Proteins--biosynthesis
Receptor Protein-Tyrosine Kinases--genetics
Recombinant Fusion Proteins--genetics
Reverse Transcriptase Polymerase Chain Reaction
Tandem Repeat Sequences
Trans-Activators--biosynthesis
Transcription, Genetic
Transfection
fms-Like Tyrosine Kinase 3