Albanese, Patricia <br/><br/>
Forced expression of p21 in GPIIb-p21 transgenic mice induces abnormalities in the proliferation of erythroid and megakaryocyte progenitors and primitive hematopoietic cells.  [electronic resource] 

 -  Experimental hematology  Nov 2002 
 - 1263-72 p.  digital 
<br/><br/> Publication Type: Comparative Study; Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0301-472X 
<br/><br/><label>Standard No.: </label>10.1016/s0301-472x(02)00933-5  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Blood Cell Count<br/>Cell Differentiation<br/>Cell Division<br/>Cell Lineage<br/>Colony-Forming Units Assay<br/>Cyclin-Dependent Kinase Inhibitor p21<br/>Cyclins--biosynthesis<br/>DNA, Complementary--genetics<br/>Erythroid Precursor Cells--cytology<br/>Gene Expression Regulation<br/>Genes, Synthetic<br/>Hematopoiesis--genetics<br/>Hematopoietic Stem Cells--cytology<br/>Homeostasis<br/>Humans<br/>Megakaryocytes--cytology<br/>Mice<br/>Mice, Transgenic<br/>Platelet Membrane Glycoprotein IIb--genetics<br/>Promoter Regions, Genetic<br/>Recombinant Fusion Proteins--physiology