DiPetrillo, Keith <br/><br/>
Urinary tumor necrosis factor contributes to sodium retention and renal hypertrophy during diabetes.  [electronic resource] 

 -  American journal of physiology. Renal physiology  Jan 2003 
 - F113-21 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S. 
<br/><br/><label>ISSN: </label>1931-857X 
<br/><br/><label>Standard No.: </label>10.1152/ajprenal.00026.2002  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Albuminuria--metabolism<br/>Animals<br/>Anti-Inflammatory Agents, Non-Steroidal--pharmacology<br/>Antigens, CD--metabolism<br/>Biomarkers<br/>Cell Separation<br/>Diabetes Mellitus, Experimental--metabolism<br/>Diabetic Nephropathies--metabolism<br/>Etanercept<br/>Hypertrophy<br/>Immunoglobulin G--pharmacology<br/>Kidney--metabolism<br/>Kidney Tubules, Distal--cytology<br/>Male<br/>Organ Size<br/>Rats<br/>Rats, Sprague-Dawley<br/>Receptors, Tumor Necrosis Factor--metabolism<br/>Receptors, Tumor Necrosis Factor, Type I<br/>Receptors, Tumor Necrosis Factor, Type II<br/>Sodium--metabolism<br/>Tumor Necrosis Factor-alpha--antagonists & inhibitors