Ruby, Carl E <br/><br/>
2,3,7,8-Tetrachlorodibenzo-p-dioxin suppresses tumor necrosis factor-alpha and anti-CD40-induced activation of NF-kappaB/Rel in dendritic cells: p50 homodimer activation is not affected.  [electronic resource] 

 -  Molecular pharmacology  Sep 2002 
 - 722-8 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, U.S. Gov't, P.H.S. 
<br/><br/><label>ISSN: </label>0026-895X 
<br/><br/><label>Standard No.: </label>10.1124/mol.62.3.722  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Antibodies--immunology<br/>Binding Sites<br/>CD40 Antigens--immunology<br/>Cell Nucleus--drug effects<br/>Cells, Cultured<br/>DNA-Binding Proteins--metabolism<br/>Dendritic Cells--drug effects<br/>I-kappa B Proteins<br/>Mice<br/>Mice, Inbred C57BL<br/>NF-KappaB Inhibitor alpha<br/>NF-kappa B--metabolism<br/>Oncogene Proteins v-rel--metabolism<br/>Peptide Hydrolases--metabolism<br/>Phosphorylation--drug effects<br/>Polychlorinated Dibenzodioxins--pharmacology<br/>Protein Binding<br/>Receptors, Aryl Hydrocarbon--metabolism<br/>Teratogens--pharmacology<br/>Transcription Factor RelA<br/>Transfection<br/>Tumor Necrosis Factor-alpha--metabolism