Sun, Ping <br/><br/>
Ganglioside loss promotes survival primarily by activating integrin-linked kinase/Akt without phosphoinositide 3-OH kinase signaling.  [electronic resource] 

 -  The Journal of investigative dermatology  Jul 2002 
 - 107-17 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, U.S. Gov't, P.H.S. 
<br/><br/><label>ISSN: </label>0022-202X 
<br/><br/><label>Standard No.: </label>10.1046/j.1523-1747.2002.01802.x  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Apoptosis--physiology<br/>Carcinoma, Squamous Cell<br/>Carrier Proteins--metabolism<br/>Caspase 9<br/>Caspases--metabolism<br/>Cell Survival--physiology<br/>Cyclin D1--genetics<br/>Cyclin E--genetics<br/>DNA, Complementary<br/>Fibronectins--metabolism<br/>G1 Phase--physiology<br/>Gangliosides--metabolism<br/>Humans<br/>Integrin beta1--metabolism<br/>Keratinocytes--cytology<br/>Neuraminidase<br/>Phosphatidylinositol 3-Kinases--metabolism<br/>Phosphorylation<br/>Protein Serine-Threonine Kinases--genetics<br/>Proto-Oncogene Proteins--genetics<br/>Proto-Oncogene Proteins c-akt<br/>S Phase--physiology<br/>Serine--metabolism<br/>Signal Transduction--physiology<br/>Skin Neoplasms<br/>Threonine--metabolism<br/>Transfection<br/>Tumor Cells, Cultured<br/>bcl-Associated Death Protein