Raeburn, Christopher D <br/><br/>
ICAM-1 and VCAM-1 mediate endotoxemic myocardial dysfunction independent of neutrophil accumulation.  [electronic resource] 

 -  American journal of physiology. Regulatory, integrative and comparative physiology  Aug 2002 
 - R477-86 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, U.S. Gov't, P.H.S. 
<br/><br/><label>ISSN: </label>0363-6119 
<br/><br/><label>Standard No.: </label>10.1152/ajpregu.00034.2002  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Antibodies--pharmacology<br/>Antineoplastic Agents, Phytogenic--pharmacology<br/>Cell Count<br/>Disease Models, Animal<br/>Disease Progression<br/>Endotoxemia--chemically induced<br/>Fluorescent Antibody Technique<br/>Heart--drug effects<br/>In Vitro Techniques<br/>Intercellular Adhesion Molecule-1--genetics<br/>Lipopolysaccharides<br/>Male<br/>Mice<br/>Mice, Inbred C57BL<br/>Mice, Knockout<br/>Myocardium--metabolism<br/>Neutrophil Infiltration--drug effects<br/>Vascular Cell Adhesion Molecule-1--metabolism<br/>Vinblastine--pharmacology