Cripe, L D <br/><br/>
Role for c-jun N-terminal kinase in treatment-refractory acute myeloid leukemia (AML): signaling to multidrug-efflux and hyperproliferation.  [electronic resource] 

 -  Leukemia  May 2002 
 - 799-812 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.; Research Support, U.S. Gov't, P.H.S. 
<br/><br/><label>ISSN: </label>0887-6924 
<br/><br/><label>Standard No.: </label>10.1038/sj.leu.2402457  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>ATP Binding Cassette Transporter, Subfamily B, Member 1--metabolism<br/>Acute Disease<br/>Bone Marrow Cells--pathology<br/>Cell Division<br/>Daunorubicin<br/>Drug Resistance, Multiple--physiology<br/>Drug Resistance, Neoplasm--physiology<br/>HL-60 Cells<br/>Humans<br/>JNK Mitogen-Activated Protein Kinases<br/>Leukemia, Myeloid--drug therapy<br/>Mitogen-Activated Protein Kinases--metabolism<br/>Phosphorylation<br/>Proto-Oncogene Proteins c-jun--metabolism<br/>Signal Transduction