Prozialeck, Walter C <br/><br/>
Chlamydia trachomatis disrupts N-cadherin-dependent cell-cell junctions and sequesters beta-catenin in human cervical epithelial cells.  [electronic resource] 

 -  Infection and immunity  May 2002 
 - 2605-13 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, U.S. Gov't, P.H.S. 
<br/><br/><label>ISSN: </label>0019-9567 
<br/><br/><label>Standard No.: </label>10.1128/IAI.70.5.2605-2613.2002  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Apoptosis<br/>Cadherins--metabolism<br/>Cervix Uteri--pathology<br/>Chlamydia trachomatis--pathogenicity<br/>Cytoskeletal Proteins--metabolism<br/>Female<br/>HeLa Cells<br/>Humans<br/>Inclusion Bodies--metabolism<br/>Intercellular Junctions--pathology<br/>Trans-Activators<br/>Uterine Cervical Neoplasms--etiology<br/>beta Catenin