Coupling endoplasmic reticulum stress to the cell death program. An Apaf-1-independent intrinsic pathway. [electronic resource]
- The Journal of biological chemistry Jun 2002
- 21836-42 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.; Research Support, U.S. Gov't, P.H.S.
0021-9258
10.1074/jbc.M202726200 doi
3T3 Cells Animals Anticarcinogenic Agents--pharmacology Apoptosis Apoptotic Protease-Activating Factor 1 Blotting, Western Brefeldin A--pharmacology Calcium--metabolism Carrier Proteins--biosynthesis Caspase 12 Caspase 9 Caspases--genetics Cell Death Cell-Free System Cells, Cultured Endoplasmic Reticulum--metabolism Endoplasmic Reticulum Chaperone BiP Enzyme Inhibitors--pharmacology Heat-Shock Proteins Mice Molecular Chaperones--biosynthesis Mutation Plasmids--metabolism Poly Adenosine Diphosphate Ribose--metabolism Precipitin Tests Protein Binding Protein Biosynthesis Protein Structure, Tertiary Protein Synthesis Inhibitors--pharmacology Proteins--metabolism Subcellular Fractions Tamoxifen--pharmacology Thapsigargin--pharmacology Time Factors