Dong, Chunming <br/><br/>
Deficient Smad7 expression: a putative molecular defect in scleroderma.  [electronic resource] 

 -  Proceedings of the National Academy of Sciences of the United States of America  Mar 2002 
 - 3908-13 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Retracted Publication 
<br/><br/><label>ISSN: </label>0027-8424 
<br/><br/><label>Standard No.: </label>10.1073/pnas.062010399  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Adult<br/>Animals<br/>Blotting, Western<br/>DNA-Binding Proteins--deficiency<br/>Down-Regulation--drug effects<br/>Enzyme-Linked Immunosorbent Assay<br/>Female<br/>Fibroblasts--drug effects<br/>Humans<br/>Immunohistochemistry<br/>Male<br/>Mice<br/>Mice, SCID<br/>Middle Aged<br/>Phosphorylation--drug effects<br/>Plasminogen Activator Inhibitor 1--analysis<br/>RNA, Messenger--genetics<br/>Scleroderma, Systemic--genetics<br/>Signal Transduction--drug effects<br/>Skin Transplantation<br/>Smad3 Protein<br/>Smad7 Protein<br/>Trans-Activators--deficiency<br/>Transcription, Genetic--drug effects<br/>Transforming Growth Factor beta--pharmacology<br/>Up-Regulation--drug effects