Tian, Hui <br/><br/>
Radiation-induced phosphorylation of Chk1 at S345 is associated with p53-dependent cell cycle arrest pathways.  [electronic resource] 

 -  Neoplasia (New York, N.Y.)   
 - 171-80 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, U.S. Gov't, P.H.S. 
<br/><br/><label>ISSN: </label>1522-8002 
<br/><br/><label>Standard No.: </label>10.1038/sj.neo.7900219  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>14-3-3 Proteins<br/>Alkaloids--pharmacology<br/>Antineoplastic Agents--pharmacology<br/>Blotting, Western<br/>Cell Cycle--drug effects<br/>Checkpoint Kinase 1<br/>Colonic Neoplasms--drug therapy<br/>Colony-Forming Units Assay<br/>Cyclin-Dependent Kinase Inhibitor p21<br/>Cyclins--metabolism<br/>DNA--radiation effects<br/>DNA Damage<br/>Drug Resistance<br/>Enzyme Inhibitors--pharmacology<br/>Humans<br/>Mutation--genetics<br/>Phosphorylation<br/>Protein Kinases--metabolism<br/>Signal Transduction<br/>Staurosporine--analogs & derivatives<br/>Tumor Cells, Cultured--drug effects<br/>Tumor Suppressor Protein p53--metabolism<br/>Tyrosine 3-Monooxygenase--metabolism