Zingarelli, Basilia <br/><br/>
Absence of inducible nitric oxide synthase modulates early reperfusion-induced NF-kappaB and AP-1 activation and enhances myocardial damage.  [electronic resource] 

 -  FASEB journal : official publication of the Federation of American Societies for Experimental Biology  Mar 2002 
 - 327-42 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, U.S. Gov't, P.H.S. 
<br/><br/><label>ISSN: </label>1530-6860 
<br/><br/><label>Standard No.: </label>10.1096/fj.01-0533com  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Apoptosis<br/>Cardiotonic Agents--metabolism<br/>Cytokines--biosynthesis<br/>HSP70 Heat-Shock Proteins--metabolism<br/>JNK Mitogen-Activated Protein Kinases<br/>Kinetics<br/>Mice<br/>Mice, Inbred C57BL<br/>Mice, Knockout<br/>Mitogen-Activated Protein Kinases--metabolism<br/>Models, Biological<br/>Myocardial Infarction--enzymology<br/>Myocardial Reperfusion Injury--enzymology<br/>Myocardium--metabolism<br/>NF-kappa B--metabolism<br/>Neutrophil Infiltration<br/>Nitric Oxide--physiology<br/>Nitric Oxide Synthase--genetics<br/>Nitric Oxide Synthase Type II<br/>Signal Transduction<br/>Transcription Factor AP-1--metabolism<br/>Tyrosine--analogs & derivatives