Chiao, Eric <br/><br/>
Overgrowth of a mouse model of the Simpson-Golabi-Behmel syndrome is independent of IGF signaling.  [electronic resource] 

 -  Developmental biology  Mar 2002 
 - 185-206 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S. 
<br/><br/><label>ISSN: </label>0012-1606 
<br/><br/><label>Standard No.: </label>10.1006/dbio.2001.0554  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Abnormalities, Multiple--genetics<br/>Animals<br/>Disease Models, Animal<br/>Female<br/>Gene Deletion<br/>Gene Expression Regulation<br/>Glypicans<br/>Heparan Sulfate Proteoglycans--genetics<br/>Insulin-Like Growth Factor I--genetics<br/>Insulin-Like Growth Factor II--genetics<br/>Male<br/>Mice<br/>Mice, Knockout<br/>Receptor, IGF Type 1--genetics<br/>Signal Transduction--genetics