Bartůnek, Petr
bFGF signaling and v-Myb cooperate in sustained growth of primitive erythroid progenitors. [electronic resource]
- Oncogene Jan 2002
- 400-10 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
ISSN: 0950-9232
Standard No.: 10.1038/sj.onc.1205103 doi
Subjects--Topical Terms: Adaptor Proteins, Signal Transducing Adaptor Proteins, Vesicular Transport Animals Blotting, Western Cell Differentiation--drug effects Cell Division--drug effects Cell Size--drug effects Cells, Cultured Cellular Senescence--drug effects Chick Embryo Erythrocytes--cytology Erythroid Precursor Cells--cytology Fibroblast Growth Factor 2--pharmacology Fibroblasts Flow Cytometry Gene Expression Regulation, Developmental--drug effects Hemoglobins--metabolism Isoenzymes--metabolism Mitogens--pharmacology Oncogene Proteins v-myb--metabolism Phospholipase C gamma Proteins--physiology Proto-Oncogene Proteins c-myb--metabolism RNA, Messenger--genetics Receptor Protein-Tyrosine Kinases--metabolism Receptor, Fibroblast Growth Factor, Type 1 Receptors, Fibroblast Growth Factor--metabolism Shc Signaling Adaptor Proteins Signal Transduction--drug effects Type C Phospholipases--metabolism