Nam, Seon Young
Low glucose-enhanced TRAIL cytotoxicity is mediated through the ceramide-Akt-FLIP pathway. [electronic resource]
- Oncogene Jan 2002
- 337-46 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
ISSN: 0950-9232
Standard No.: 10.1038/sj.onc.1205068 doi
Subjects--Topical Terms: Adenocarcinoma--metabolism Apoptosis--drug effects Apoptosis Regulatory Proteins CASP8 and FADD-Like Apoptosis Regulating Protein Carrier Proteins--metabolism Caspases--metabolism Cell Survival--drug effects Ceramides--metabolism Enzyme Activation--drug effects Glucose--deficiency Humans Intracellular Signaling Peptides and Proteins Male Membrane Glycoproteins--toxicity Poly(ADP-ribose) Polymerases--metabolism Prostatic Neoplasms--metabolism Protein Serine-Threonine Kinases Proto-Oncogene Proteins--metabolism Proto-Oncogene Proteins c-akt Receptors, TNF-Related Apoptosis-Inducing Ligand Receptors, Tumor Necrosis Factor--metabolism Spectrometry, Mass, Electrospray Ionization TNF-Related Apoptosis-Inducing Ligand Tumor Cells, Cultured Tumor Necrosis Factor-alpha--toxicity