Zhang, J <br/><br/>
Expression of hypoxia-inducible factors in human endometrium and suppression of matrix metalloproteinases under hypoxic conditions do not support a major role for hypoxia in regulating tissue breakdown at menstruation.  [electronic resource] 

 -  Human reproduction (Oxford, England)  Feb 2002 
 - 265-74 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0268-1161 
<br/><br/><label>Standard No.: </label>10.1093/humrep/17.2.265  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Aryl Hydrocarbon Receptor Nuclear Translocator<br/>Basic Helix-Loop-Helix Transcription Factors<br/>Blotting, Western<br/>Cell Hypoxia--physiology<br/>Cells, Cultured<br/>DNA-Binding Proteins<br/>Endometrium--cytology<br/>Endothelial Growth Factors--metabolism<br/>Female<br/>Humans<br/>Hypoxia-Inducible Factor 1, alpha Subunit<br/>Immunohistochemistry<br/>Isoenzymes--metabolism<br/>Lymphokines--metabolism<br/>Matrix Metalloproteinases--metabolism<br/>Matrix Metalloproteinases, Membrane-Associated<br/>Menstruation--physiology<br/>Metalloendopeptidases--metabolism<br/>Receptors, Aryl Hydrocarbon<br/>Stromal Cells--metabolism<br/>Trans-Activators--metabolism<br/>Transcription Factors--metabolism<br/>Vascular Endothelial Growth Factor A<br/>Vascular Endothelial Growth Factors