Wilson, J B <br/><br/>
The Chinese hamster FANCG/XRCC9 mutant NM3 fails to express the monoubiquitinated form of the FANCD2 protein, is hypersensitive to a range of DNA damaging agents and exhibits a normal level of spontaneous sister chromatid exchange.  [electronic resource] 

 -  Carcinogenesis  Dec 2001 
 - 1939-46 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0143-3334 
<br/><br/><label>Standard No.: </label>10.1093/carcin/22.12.1939  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Bleomycin--pharmacology<br/>CHO Cells<br/>Cell Line<br/>Cricetinae<br/>DNA Damage--drug effects<br/>DNA-Binding Proteins--genetics<br/>Epoxy Compounds--pharmacology<br/>Fanconi Anemia--genetics<br/>Fanconi Anemia Complementation Group D2 Protein<br/>Fanconi Anemia Complementation Group G Protein<br/>Gamma Rays<br/>Genetic Complementation Test<br/>Humans<br/>Hybrid Cells<br/>Mechlorethamine--pharmacology<br/>Mitomycin--pharmacology<br/>Mutagens--pharmacology<br/>Mutation--genetics<br/>Nuclear Proteins--chemistry<br/>Sister Chromatid Exchange--drug effects<br/>Ubiquitin--metabolism<br/>Ultraviolet Rays