Kakimi, K <br/><br/>
Blocking chemokine responsive to gamma-2/interferon (IFN)-gamma inducible protein and monokine induced by IFN-gamma activity in vivo reduces the pathogenetic but not the antiviral potential of hepatitis B virus-specific cytotoxic T lymphocytes.  [electronic resource] 

 -  The Journal of experimental medicine  Dec 2001 
 - 1755-66 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S. 
<br/><br/><label>ISSN: </label>0022-1007 
<br/><br/><label>Standard No.: </label>10.1084/jem.194.12.1755  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Chemokine CXCL10<br/>Cytotoxicity, Immunologic<br/>Hepatitis B--genetics<br/>Hepatitis B virus--immunology<br/>Interferon-gamma--genetics<br/>Liver--immunology<br/>Mice<br/>Mice, Transgenic<br/>Monokines--genetics<br/>T-Lymphocytes, Cytotoxic--immunology