Kitazawa, M <br/><br/>
Dieldrin-induced oxidative stress and neurochemical changes contribute to apoptopic cell death in dopaminergic cells.  [electronic resource] 

 -  Free radical biology & medicine  Dec 2001 
 - 1473-85 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S. 
<br/><br/><label>ISSN: </label>0891-5849 
<br/><br/><label>Standard No.: </label>10.1016/s0891-5849(01)00726-2  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>3,4-Dihydroxyphenylacetic Acid--analysis<br/>Animals<br/>Apoptosis--drug effects<br/>Cell Survival--drug effects<br/>Chromatography, High Pressure Liquid<br/>Dieldrin--administration & dosage<br/>Dopamine--metabolism<br/>Dose-Response Relationship, Drug<br/>Enzyme Inhibitors--pharmacology<br/>Flow Cytometry<br/>Intracellular Membranes--drug effects<br/>L-Lactate Dehydrogenase--analysis<br/>Lipid Peroxidation<br/>Membrane Potentials--drug effects<br/>Mitochondria--drug effects<br/>Monoamine Oxidase Inhibitors--pharmacology<br/>Oxidative Stress--drug effects<br/>PC12 Cells<br/>Parkinson Disease, Secondary--chemically induced<br/>Rats<br/>Reactive Oxygen Species--metabolism<br/>Superoxide Dismutase--pharmacology<br/>Tyrosine 3-Monooxygenase--antagonists & inhibitors