O'Brien, N C <br/><br/>
Inhibition of nitric oxide synthase initiates relapsing remitting experimental autoimmune encephalomyelitis in rats, yet nitric oxide appears to be essential for clinical expression of disease.  [electronic resource] 

 -  Journal of immunology (Baltimore, Md. : 1950)  Nov 2001 
 - 5904-12 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0022-1767 
<br/><br/><label>Standard No.: </label>10.4049/jimmunol.167.10.5904  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Arginine--analogs & derivatives<br/>Cells, Cultured<br/>Encephalomyelitis, Autoimmune, Experimental--enzymology<br/>Enzyme Inhibitors--pharmacology<br/>Kinetics<br/>Lymph Nodes--immunology<br/>Lymphocyte Activation<br/>Macrophages--immunology<br/>Myelin Basic Protein--immunology<br/>Nitric Oxide--physiology<br/>Nitric Oxide Synthase--antagonists & inhibitors<br/>Nitric Oxide Synthase Type II<br/>Rats<br/>Rats, Inbred Lew<br/>Reactive Nitrogen Species--blood<br/>Recurrence<br/>Spinal Cord--metabolism