Schipani, E <br/><br/>
Hypoxia in cartilage: HIF-1alpha is essential for chondrocyte growth arrest and survival.  [electronic resource] 

 -  Genes & development  Nov 2001 
 - 2865-76 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, U.S. Gov't, P.H.S. 
<br/><br/><label>ISSN: </label>0890-9369 
<br/><br/><label>Standard No.: </label>10.1101/gad.934301  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Alleles<br/>Animals<br/>Bone and Bones--abnormalities<br/>Bromodeoxyuridine--metabolism<br/>Cartilage--metabolism<br/>Cell Division<br/>Cell Survival<br/>Chondrocytes--metabolism<br/>Endothelial Growth Factors--metabolism<br/>Hypertrophy<br/>Hypoxia<br/>Hypoxia-Inducible Factor 1, alpha Subunit<br/>Immunohistochemistry<br/>In Situ Hybridization<br/>In Situ Nick-End Labeling<br/>Integrases--metabolism<br/>Lymphokines--metabolism<br/>Mice<br/>Mice, Knockout<br/>Mice, Transgenic<br/>Oxygen--metabolism<br/>Phenotype<br/>Polypyrimidine Tract-Binding Protein<br/>RNA-Binding Proteins--metabolism<br/>Ribonucleoproteins--metabolism<br/>Time Factors<br/>Trachea--abnormalities<br/>Transcription Factors--metabolism<br/>Up-Regulation<br/>Vascular Endothelial Growth Factor A<br/>Vascular Endothelial Growth Factors<br/>Viral Proteins--metabolism